ADVENT is a non-promotional global medical education program by Sanofi and Regeneron, designed to engage healthcare professionals (HCPs) and advance the understanding of the role of type 2 inflammation across several diseases within dermatology, pulmonology, rhinology, and gastroenterology. Offering a wide variety of face-to-face and digital opportunities, the ADVENT program aims to provide HCPs with high-quality, fair-balanced, and continuous education to advance clinical management and improve patient care. At its core, the objectives of the ADVENT program are to educate on type 2 inflammation, with a focus on advancing understanding of pathophysiology and highlighting the significant burden suffered by patients, families, and caregivers. The ADVENT program prioritizes the exploration and dissemination of evolving data on new therapeutic approaches to management and prompts understanding of the multidisciplinary implications and cumulative burden associated with type 2 inflammatory diseases, including atopic dermatitis, prurigo nodularis, chronic spontaneous urticaria, and bullous pemphigoid.
The name ADVENT, formed by combining ‘AD’ (atopic dermatitis & dermatology), ‘VENT’ (ventilation = pulmonology), and ‘ENT’ (rhinology), signifies a novel and innovative approach to medical education, which has historically relied on traditional, short-term IME/CME programs. The ADVENT program was designed to disrupt these conventional formats, offering a continuously evolving curriculum of varied digital resources and in-person experiences conducive for diverse learning styles, all of which are developed in collaboration with over 100 global medical experts.
Sanofi and Regeneron are global leaders in pharmaceutical development, each with a long history of supporting frontline physicians to improve human health and reduce the burden of disease. Driven by the same commitment to patients and their families, Sanofi and Regeneron have partnered to take on type 2 inflammation and the range of chronic conditions in which excessive inflammation plays a role.